Integrated and holistic healthcare clinic London

Artemisinin IV Infusions

Artemisinin is a promising novel-anticancer therapy now available as an IV drip from our clinic in London

What is Artemisinin

Artemisinin is a compound extracted from the wormwood plant, Artemisia annua L.

This active principle has been used for centuries as part of the treatment for malaria, fevers and inflammation. Researchers at N.C.I.’s (National Cancer Institute) Developmental Therapeutic Program discovered that artemisinin may also be effective not only in destroying parasites but also cancer cells, which store iron in high concentrations.

Artemisinin and Cancer

Artemisinin has been found to hold unique properties which selectively attack cancer cells while leaving normal and healthy cells undamaged. It is activated by iron, which cancer cells accumulate as it is crucial for their rapid growth and proliferation. Most cancer cells have high rates of iron intake and express a high concentration of transferrin receptors on the cell surface. There seems to be a correlation between the aggressiveness of the tumour and the number of these iron receptors.

Artemisinin has a peroxide bond that is activated by iron in order to generate hydrogen peroxide. This free radical of Oxygen stresses cancer cells, which are deficient in peroxide-neutralizing catalase enzyme. Normal cells with catalase can harmlessly dissipate the peroxides. Healthy cells are approximately 100 times less vulnerable to dying from artemisinin than cancer cells. 

Researchers at N.C.I. found artemisinin to have anti-cancer activity against 55 cancer cell lines.

Most importantly, Artemisinin has shown to have an effect in leukaemia, B-cell lymphoma cells, colorectal cancer, thyroid cancer, lung and breast cancer (including radiation resistant breast cancer).

Artemisinin has also shown some effect against cancers that were traditionally resistant to chemotherapy. Other cancers that it was found beneficial include melanoma, ovarian, prostate, renal and central nervous system cancers such as glioblastoma and neuroblastoma.

Clinical studies on patients with breast, cervical, liver and lung cancer have shown tumour reductions of up to 70%, a short-term increase in lifespan and even remission.

About Artemisinin

Artemisia annua, which is often known as sweet sagewort or wormwood,  has been used in traditional Chinese Medicine for malaria, fevers, inflammation, bleeding and headaches. In vitro studies show that artemisinin, which is the active principle of artemisia annua, may be an effective way of treating African sleeping sickness and protozoal infections and chagas disease ( 9). Cytotoxic effects of A. annua compounds have also been evaluated in tumour cell lines (1) (18) (19) (20) (25) (26). Artemisinin-based combination therapies are part of the standard treatment used for malaria. Reviews have indicated that it is as effective as quinine (4) (5), but that its uses may be limited because of an increased risk of relapse (6) (7). It is also unclear whether it is effective against quinine-resistant malaria strains. Additional drug development is being pursued after reports of artemisinin-based therapy resistance that are also appearing (28).


There are limited studies artemisia for other health conditions. In one RCT, a low-dose artemisia formulation shows clinically relevant pain reductions in patients with hip or knee osteoarthritis (3). A recent study has indicated long term safety, with improvements maintained at 6 months. Studies in advanced cancer patients have suggested that oral add-on artesunate, an artemisinin derivative, is well tolerated, although ototoxicity monitoring is needed (13) (21). Different studies showed that oral artesunate did not produce a response (29) (30), although modest clinical activity was seen with intravenous administration (30). Further research is needed to determine the conditions under which compounds derived from artemisia may be safe and effective.


The active component of A. annua, Artemisinin, makes use of its antimalarial effects by free radicals formed via cleavage of the endoperoxide bond in its structure, which are responsible for eradicating the Plasmodium species (23). It also induces apoptosis and cell cycle arrest of Leishmani donovani promastigotes (8), has antiproliferative effects on medullary thyroid carcinoma cells (2), and by modulating p38 and calcium signalling (14), 

induces apoptosis in a lung cancer cell line.


Research has indicated that it significantly inhibited cell growth and proliferation, and caused G1 cell-cycle arrest in neuroblastoma cell lines (25). Dihydroartemisinin, a semisynthetic derivative of artemisinin, demonstrates anti-inflammatory activity by attenuating COX-2 production via downregulation of serine/threonine kinase and MAPK pathways  (24). Recent findings suggest that dihydroartemisinin-triggered apoptosis in colorectal cells occurs through the ROS-mediated mitochondria-dependent pathway (26).


Artemisinin works as a hormone regulator, specifically reducing excess prolactin and oestrogen in breast cancer. It is activated by ionic ferrous iron, which cancer cells accumulate. Iron is an essential compound for cancer cell proliferation. Most cancer cells have high rates of iron intake and express a high concentration of transferrin receptors on the cell surface. The rapid growth of abnormal cells utilise relatively large amounts of iron, mainly in the form of holotransferrin.


Dihydro-artemisinin has a peroxide bond that is activated by iron in order to generate hydrogen peroxide. This free radical of Oxygen stresses cancer cells, which are always deficient in peroxide-neutralizing catalase enzyme. Normal cells with catalase can harmlessly dissipate the peroxides. Healthy cells are approximately 100 times less vulnerable to dying from artemisinin than cancer cells. In cancer cells, the high-valent oxo-iron species create a surge of endoperoxides, depolarizing the mitochondrial membranes and cause a disruption in the electron transport chain.


Other important actions of artemisinin:


  • Essential in the regulation of P53 DNA repair gene and cyclin dependent kinases
  • Induces apoptosis and slows growth in cancers such as fibrosarcoma, lymphoma, breast, pancreatic, oesophageal, prostate, and ovarian/fallopian/peritoneal carcinomas, squamous cell carcinomas and liver hepatocellular cancer
  • Artemisinin eases down-regulation of nuclear factor kappa B, which is the master control gene for inflammation. A sign of intense inflammation in the cancer cell is high blood levels of CRP and ESR, and also mid- to high-normal LDH. These markers can help to indicate which patients are good candidates for artemisinin therapy.
  • Aids in the inhibiting angiogenesis , which disrupts the blood supply to tumours.
  • Regulates important epigenetics or DNA silencing, including methylation and histone protein acetylation.
  • Targets translationally controlled tumour protein TCTP
  • inhibits cysteine protease enzyme, and also a SERCA-type calcium transporter enzyme.



What makes the properties of artemisinin and its derivatives integral components of complementary cancer therapy are its antineoplastic properties. While being tested by the National Cancer Institute in the USA, specifically for it anti-cancer activity on 55 of the most common  cancer cell lines, the study (23)  showed breast, prostate, ovary, colon, kidney, CNS and melanoma cells showed an increased Artesunate sensitivity. Further research noted a specific sensitivity to diffuse large B-cell lymphoma cells [24,25]. Clinical studies on patients with breast, cervical, liver and lung cancer have shown tumor reductions of up to 70%, a short-term increase in lifespan and even remission [26-28].



Artemisinin derivatives also showed synergistic effects with chemotherapeutic agents. When used in combination with gemcitabine in pancreatic carcinoma, growth inhibition was increased fourfold in vitro and in vivo, with the apoptosis rate also being doubled compared to monotherapy with gemcitabine [29]. An additional study indicated increased inhibition of metastasis and cancer cell growth in murine Lewis lung carcinoma cell lines when used in conjunction with cyclophosphamide compared to monotherapy [30].

Cancer cells are often characterized by developed resistance to chemotherapeutic agents. A combination of artesunate with doxorubicin and pirarubicin showed increased cytotoxicity in K562/ADR leukaemia and GLC4/ADR lung carcinoma cells [31].

Along with the above , sensitization was also seen in combination with cisplatin in ovarian carcinoma [32]. Even independent of p53 status, artemisinin was capable of inducing  strong sensitization to gemcitabine in hepatocellular carcinoma cells [33].


Due to artemisinin and its derivatives' ability to selectively disrupt tumour growth, its cell cycle and proliferation pathways [34], it has an anticarcinogenic effect. This process can be traced back  to the particular properties of the tumour cells, such as increased metabolism, increased blood flow and thus increased iron and transferrin levels.

In conjunction to being well tolerated, artemisinin derivatives are also known to have an antineoplastic effect.

The anti-carcinogenic effect is based on the structural endoperoxide bridge, which can form cytotoxic radicals with heme groups or intracellular iron. These radicals primarily lead to cell cycle arrest and disrupt proliferative pathways [35-37].

The reason for this specific effect against cancer cells is due to their increased metabolism,  their increased requirement for iron and their increased number of transferrin receptors  when compared to normal cells [38, 39-42].


In addition,  artemisinin and its derivatives were able to induce various cellular signalling pathways that led to apoptosis or necrosis in gastric and oesophageal tumour cell lines [44-46,47]. Along with this, artemisinin derivatives showed a reduction in the risk of metastasis by increasing cell adhesion [48].


Adverse effects 

Case reports

  •                 Hepatitis: In a 52-year-old man following consumption of a herbal supplement containing artemisinin (17).
  •                 Acute cholestatic hepatitis: In a patient due to ingestion of artemisia tea as prophylaxis against malaria (31).
  •                 Delayed hemolytic anaemia: Two cases after either oral or intravenous therapy with artemisinin-based treatment for malaria (32) (33). It is thought this reaction may be related to higher parasite loads (32).
  •                 Ototoxicity and vertigo: Possibly related to oral artesunate, an active artemisia compound, among several advanced breast cancer patients in a safety trial (13) (21). The study drug was otherwise largely well tolerated among patients.
  •                 Dermatitis: With topical use of artemisia (11).



Drug interactions

CYP450 substrates: In laboratory studies, artemisia extracts induced CYP2B6 and CYP3A4 (27) and may affect the serum concentration of drugs metabolized by these enzymes. Clinical relevance has yet to be determined.




  1.             Singh NP, Lai HC. Artemisinin induces apoptosis in human cancer cells. Anticancer Res. 2004 Jul-Aug;24(4):2277-80.
  2.                 Zheng GQ. Cytotoxic terpenoids and flavonoids from Artemisia annua. Planta Med. 1994 Feb;60(1):54-7.
  3.                 Stebbings S, Beattie E, McNamara D, et al. A pilot randomized, placebo-controlled clinical trial to investigate the efficacy and safety of an extract of Artemisia annua administered over 12 weeks, for managing pain, stiffness, and functional limitation associated with osteoarthritis of the hip and knee. Clin Rheumatol. Jul 2016;35(7):1829-1836.
  4.                 McIntosh HM, Olliaro P. Artemisinin derivatives for treating severe malaria. Cochrane.Database.Syst.Rev. 2000;CD000527.
  5.                 McIntosh HM, Olliaro P. Artemisinin derivatives for treating uncomplicated malaria. Cochrane.Database.Syst.Rev. 2000;CD000256.
  6.                 Mueller MS, et al. Randomized controlled trial of a traditional preparation of Artemisia annua L. (Annual Wormwood) in the treatment of malaria. Trans R Soc Trop Med Hyg. 2004;98:318-21.
  7.                 Blanke CH, Naisabha GB, Balema MB, et al. Herba Artemisiae annuae tea preparation compared to sulfadoxine-pyrimethamine in the treatment of uncomplicated falciparum malaria in adults: a randomized double-blind clinical trial. Trop Doct. Apr 2008;38(2):113-116.
  8.                 Sen R, Bandyopadhyay S, Dutta A, et al. Artemisinin triggers induction of cell-cycle arrest and apoptosis in Leishmania donovani promastigotes. J Med Microbiol. Sep 2007;56(Pt 9):1213-1218.
  9.                 Mishina YV, Krishna S, Haynes RK, Meade JC. Artemisinins inhibit Trypanosoma cruzi and Trypanosoma brucei rhodesiense in vitro growth. Antimicrob Agents Chemother. May 2007;51(5):1852-1854.
  10.                 Hunt S, Stebbings S, McNamara D. An open-label six-month extension study to investigate the safety and efficacy of an extract of Artemisia annua for managing pain, stiffness and functional limitation associated with osteoarthritis of the hip and knee. N Z Med J. Oct 28 2016;129(1444):97-102.
  11.                 Skyles AJ, Sweet BV. Alternative therapies. Wormwood. Am J Health Syst.Pharm. 2004;61:239-42.
  12.                 Rinner B, et al. Activity of novel plant extracts against medullary thyroid carcinoma cells. Anticancer Res 2004;24:495-500.
  13.                 Konig M, von Hagens C, Hoth S, et al. Investigation of ototoxicity of artesunate as add-on therapy in patients with metastatic or locally advanced breast cancer: new audiological results from a prospective, open, uncontrolled, monocentric phase I study. Cancer Chemother Pharmacol. Feb 2016;77(2):413-427.
  14.                 Mu D, Zhang W, Chu D, et al. The role of calcium, P38 MAPK in dihydroartemisinin-induced apoptosis of lung cancer PC-14 cells. Cancer Chemother Pharmacol. Apr 2008;61(4):639-645.
  15.                 Rath K, et al. Pharmacokinetic study of artemisinin after oral intake of a traditional preparation of Artemisia annua L. (annual wormwood). Am J Trop Med Hyg. 2004;70:128-32.
  16.                 Payne AG. Exploiting intracellular iron and iron-rich compounds to effect tumor cell lysis. Med Hypotheses 2003;61:206-9.
  17.                 Centers for Disease Control and Prevention. Hepatitis Temporally Associated with an Herbal Supplement Containing Artemisinin —- Washington, 2008. Accessed December 10, 2021.
  18.                 Lai H, Nakase I, Lacoste E, Singh NP, Sasaki T. Artemisinin-transferrin conjugate retards growth of breast tumors in the rat. Anticancer Res. 2009 Oct;29(10):3807-10.
  19.                 Willoughby JA Sr, Sundar SN, Cheung M, et al. Artemisinin blocks prostate cancer growth and cell cycle progression by disrupting Sp1 interactions with the cyclin-dependent kinase-4 (CDK4) promoter and inhibiting CDK4 gene expression. J Biol Chem. 2009 Jan 23;284(4):2203-13.
  20.                 Zhai DD, Supaibulwatana K, Zhong JJ. Inhibition of tumor cell proliferation and induction of apoptosis in human lung carcinoma 95-D cells by a new sesquiterpene from hairy root cultures of Artemisia annua. Phytomedicine. 2010 Sep;17(11):856-61.
  21.                 Konig M, von Hagens C, Hoth S, et al. Erratum to: Investigation of ototoxicity of artesunate as add-on therapy in patients with metastatic or locally advanced breast cancer: new audiological results from a prospective, open, uncontrolled, monocentric phase I study. Cancer Chemother Pharmacol. Jun 2016;77(6):1321.
  22.                 van der Kooy F, Sullivan SE. The complexity of medicinal plants: the traditional Artemisia annua formulation, current status and future perspectives.J Ethnopharmacol. 2013 Oct 28;150(1):1-13.
  23.                 Ho WE, Peh HY, Chan TK, Wong WS. Artemisinins: pharmacological actions beyond anti-malarial. Pharmacol Ther. 2014 Apr;142(1):126-39.
  24.                 Kim HG, Yang JH, Han EH, et al. Inhibitory effect of dihydroartemisinin against phorbol ester-induced cyclooxygenase-2 expression in macrophages.  Food Chem Toxicol. 2013 Jun;56:93-9.
  25.                 Zhu S, Liu W, Ke X, et al. Artemisinin reduces cell proliferation and induces apoptosis in neuroblastoma. Oncol Rep. 2014 Sep;32(3):1094-100.
  26.                 Lu M, Sun L, Zhou J, Yang J.Dihydroartemisinin induces apoptosis in colorectal cancer cells through the mitochondria-dependent pathway. Tumour Biol. 2014 Jun;35(6):5307-14.
  27.                 Xing J, Kirby BJ, Whittington D, et al. Evaluation of P450 inhibition and induction by artemisinin antimalarials in human liver microsomes and primary human hepatocytes. Drug Metab Dispos. 2012 Sep;40(9):1757-64.
  28.                 van der Pluijm RW, Tripura R, Hoglund RM, et al. Triple artemisinin-based combination therapies versus artemisinin-based combination therapies for uncomplicated Plasmodium falciparum malaria: a multicentre, open-label, randomised clinical trial. Lancet. Apr 25 2020;395(10233):1345-1360.
  29.                 von Hagens C, Walter-Sack I, Goeckenjan M, et al. Long-term add-on therapy (compassionate use) with oral artesunate in patients with metastatic breast cancer after participating in a phase I study (ARTIC M33/2). Phytomedicine. Feb 15 2019;54:140-148.
  30.                 Deeken JF, Wang H, Hartley M, et al. A phase I study of intravenous artesunate in patients with advanced solid tumor malignancies. Cancer Chemother Pharmacol. Mar 2018;81(3):587-596.
  31.                 Ruperti-Repilado FJ, Haefliger S, Rehm S, et al. Danger of Herbal Tea: A Case of Acute Cholestatic Hepatitis Due to Artemisia annua Tea. Front Med (Lausanne). 2019;6:221.
  32.                 Conlon CC, Stein A, Colombo RE, et al. Post-artemisinin delayed hemolysis after oral therapy for P. falciparum infection. IDCases. 2020;20:e00741.
  33.                 Hasegawa C, Kudo M, Maruyama H, et al. Severe delayed haemolytic anaemia associated with artemether-lumefantrine treatment of malaria in a Japanese traveller. J Infect Chemother. Mar 2018;24(3):216-219.


  1. Crespo-Ortiz, M.P.; Wei, M.Q. Antitumor activity of artemisinin and its derivatives: From a well-
    known antimalarial agent to a potential anticancer drug. J. Biomed. Biotechnol. 2012, 247597. 
  2. Olliaro, P.L.; Haynes, R.K.; Meunier, B.; Yuthavong, Y. Possible modes of action of the artemisinin-type compounds. Trends Parasitol. 2001, 17, 122–126.
  3. Meshnick, S.R.; Thomas, A.; Ranz, A.; Xu, C.M.; Pan, H.Z. Artemisinin (qinghaosu): The role of intra- cellular hemin in its mechanism of antimalarial action. Mol. Biochem. Parasitol. 1991, 49, 181–189.
  4. Zhang, S.; Gerhard, G.S. Heme activates artemisinin more efficiently than hemin, inorganic
    iron, or hemoglobin. Bioorg. Med. Chem. 2008, 16, 7853–7861.
  5. Lai, H.C.; Singh, N.P.; Sasaki, T. Development of artemisinin compounds for cancer treatment.
    Investog. New Drugs 2013, 31, 230–246.
  6. Reizenstein, P. Iron, free radicals and cancer. Med. Oncol. Tumor Pharmacother. 1991, 8,
  7. Lui, G.Y.L.; Kovacevic, Z.; Richardson, V.;Merlot, A.M.; Kalinowski, D.S.; Richardson, D.R.
    Targeting cancer cells by binding iron: Dissecting cellular signaling pathways. Oncotarget
    2015, 6, 18748–18779.
  8. Ward, P.S.; Thompson, C.B. Metabolic reprogramming: A cancer hallmark even Warburg did
    not anticipate. Cancer Cell. 2012, 21, 297–308.
  9. Niitsu, Y.; Kohgo, Y.; Nishisato, T.; Kondo, H.; Kato, J.; Urushizaki, Y.; Urushizaki, I. Transferrin
    receptors in human cancerous tissues. Tohoku J. Exp. Med. 1987, 153, 239–243.
  10. Hamacher-Brady, A.; Stein, H.A.; Turschner, S.; Toegel, I.; Mora, R.; Jennewein, N.; Efferth, T.; Eils, R.; Brady, N.R. Artesunate activates mitochondrial apoptosis in breast cancer cells
    via ironcatalyzed lysosomal reactive oxygen species production. J. Biol. Chem. 2011, 286,
  11. Nakase, I.; Gallis, B.; Takatani-Nakase, T.; Oh, S.; Lacoste, E.; Singh, N.P.; Goodlett, D.R.; Tanaka, S.; Futaki, S.; Lai, H.; et al. Transferrin receptor-dependent cytotoxicity of artemisinin- transferrin conjugates on prostate cancer cells and induction of apoptosis. Cancer Lett. 2009, 274, 290–298. 
  12. Mercer, A.E.; Maggs, J.L.; Sun, X.M.; Cohen, G.M.; Chadwick, J.; O’Neill, P.M.; Park, B.K. Evidence for the involvement of carbon-centered radicals in the induction of apoptotic cell death by artemisinin compounds. J. Biol. Chem. 2007, 282, 9372–9382. 
  13. Efferth, T.; Giaisi, M.; Merling, A.; Krammer, P.H.; Li-Weber, M. Artesunate induces ROS-media- ted apoptosis in doxorubicinresistant T leukemia cells. PLoS ONE 2007, 2, e693. 
  14. Liu, L.; Zuo, L.F.; Zuo, J. Artesunate induces apoptosis and inhibits growth of Eca109 and Ec9706 human esophageal cancer cell lines in vitro and in vivo. Mol. Med. Rep. 2015, 12, 1465–1472 
  15. Weifeng, T.; Feng, S.; Xiangji, L.; Changging, S.; Zhiquian, Q.; Huazhong, Z.; Peining, Y.; Yong, Y.; Mengchao, W.; Xiaoqing, J.; et al. Artemisinin inhibits in vitro and in vivo invasion and metastasis of human hepatocellular carcinoma cells. Phytomedicine 2011, 18, 158–162.

Book Your Artemisinin IV Drip

Boost Your Immunity - Feel Better.

Vitamin IV drip near me

You do not need to worry about finding an IV vitamin drip clinic nearby. Our at-home IV therapy, mobile concierge service can be with you in no time and infuse the necessary multivitamins and minerals you need at the comfort of your own home.

What is intravenous nutrient therapy?

Intravenous nutrient therapy, or IV therapy, was meant to have been started by John Myers a Physician from Baltimore who pioneered the use of administering vitamins and minerals intravenously as part of the overall treatment of various medical conditions.

Intravenous therapy is the administration of fluids, medications, or nutritional supplements that are administered directly into your vein since they are needed quickly by the body. It is a common, minimally invasive process to infuse these necessary elements directly into the bloodstream, where it can be utilised rapidly to treat a myriad of common complaints and ailments.

Where to get an iv drip in London?

You can get easily get an IV drip in our clinic in London. You can book you rehydrate and recover appointment using our online booking diary or over the phone.

Who will perform my IV Drip Therapy?

The infusion will be performed by our doctor or one of our registered nurses (RGN). Our nurses have years of experience working for the NHS and private sector, and you can therefore rest assured that you are safe and in good hands.

How much does an IV drip cost?

You will find the vitamin IV drips affordable and the cost of the drips is clearly stated with the drips descriptions. The price you see is the price you pay, with no hidden extras.

Our vitamin and mineral, rehydration therapy will deliver vital nutrients straight into your bloodstream.

How does intravenous nutrient therapy work?

IV therapy works by allowing your body to absorb 100% of the vitamins, as opposed to only 10% with oral supplements, and can leave you feeling great for weeks.

The rationale for IV administration of nutrients is that it can achieve serum concentrations that are not obtainable with oral or intramuscular administration. This is because of the more rapid entry of the nutrients into the bloodstream.

What are the benefits of intravenous nutrient therapy?

Almost anyone can benefit from intravenous nutrient therapy; whether you are dealing with the stress and strains of modern-day life, feeling run down and tired, looking to boost sports performance and recovery, as part of a dietary programme to aid weight loss or to aid detoxification protocols, the rewards are endless.

Many relatively healthy patients will also choose to come in for periodic infusions because they noticed it enhanced their overall wellbeing for periods of a week or several months.

  • Immune support
  • Dehydration
  • Sports performance
  • Fatigue (including Chronic Fatigue Syndrome)
  • Skin conditions
  • Hypothyroidism
  • Heavy metal toxicity
  • Lyme disease
  • Mould toxicity
  • Osteoporosis
  • Migraines
  • Fibromyalgia
  • Asthma
  • Chronic sinusitis
  • Heart conditions
  • Chronic urticaria
  • Dysmenorrhea
  • Multiple Sclerosis
  • Plus more…

What types of intravenous infusions are available?

Based on the research done by the renowned John Myers and many more people since then, we offer a wide range of nutritional IV's to enhance patients’ health, reduce symptoms and help reverse chronic disease.

Among the many infusions, we offer is the classic Myers cocktail, which has been used to treat patients for over 30 years. Similar to the original Myers but with increased nutrient levels, we have our bespoke immune-boosting and sports performance formulas, which are very popular. Our vitamin C and glutathione IV's are extremely effective at improving detoxification, particularly when there is heavy metal toxicity, oxidative stress and reducing pathogen loads.

We can also blend bespoke IV’s for patients as and when required.

The Most Efficient and Effective Way to Deliver Nutrients to Your Body

IV Therapy treatments can help boost your immune system, especially if youare run down, stressed out or generally feel exhausted and in need of a pick-me-up. Many patients have found it highly effective in reducing a variety of troubling symptoms including fatigue, headaches, muscle, pain brain fog, fibromyalgia and much more.

The Benefits of IV Vitamin Therapy

IV vitamin therapy is so effective as a fatigue treatment and pain reliever because it injects essential, healing substances directly into the gut. A few benefits include:

  • Increased energy
  • Enhanced mood
  • Mental clarity
  • Improved sleep
  • Decreased stress and anxiety
  • Increase immunity
  • Muscle recovery
  • Hydration

IV Vitamin Therapy is the fastest, most efficient way to deliver vitamins, amino acids, antioxidants, and other vital nutrients to your circulatory system.

Why IV Vitamin Therapy Over Oral Vitamins

When IV Vitamin Therapy is infused straight into the bloodstream via a vein, you get the benefit of 100% absorption, allowing these nutrients to be immediately available for cellular use. IV Vitamin Therapy can also safely deliver higher doses of nutritional vitamins that would not be tolerated if taken orally. When it comes to preventing illness, increasing energy, and slowing the effects of ageing, IV Vitamin Therapy from Mitchell Medical Group will give your immune system the fuel it needs to maximize your health.

One of the most powerfully effective treatments that we have found for treating chronic illnesses like chronic fatigue and fibromyalgia is the use of IV vitamin therapy.

The difference between oral vitamins and injected vitamins has to do with the gut’s ability to absorb the vitamins. Oral vitamins are swallowed and end up in the stomach, where the stomach’s acid has to do some heavy work to break down the vitamin pills and then can start to slowly absorb the nutrients. In many cases, the oral vitamins swallowed are only absorbed 30%; this can be even lower in patients taking acid blockers or who have digestive problems.

What is IV Vitamin Therapy?

IV Vitamin Therapy is a method of infusing vitamins, amino acids, and antioxidants directly into your body. The difference between oral vitamins and injected vitamins has to do with the gut’s ability to absorb the vitamins. The first IV vitamin drips were developed and administered by Dr. John Myers in the 1970s.

While you’re undergoing an IV vitamin drip, your body is receiving a higher concentration of the vitamins themselves. A vitamin that’s taken by mouth gets broken down in the stomach and digestive tract, and is limited on how much can be absorbed (50 per cent) – this can be even lower in patients taking acid blockers or who have digestive problems. In contrast, when the vitamin is given through an IV (intravenously), it’s absorbed in a much higher percentage (90 per cent) because it goes right into the body without the need for digestive help.

What’s In Our IV Vitamin Drip?

Our high dose vitamin injections incorporate key vitamins that improve the immune system and address nutrient deficiency, making it an ideal therapy for people with vitamin deficiencies. Essentially, the IV therapies (also known as Myer’s cocktails) dramatically jump-start the body’s systems, which can markedly shorten the time it takes to begin feeling better.

Our IV Vitamin Therapy Includes the Following Vitamins:

  • Vitamin C
  • B complex
  • Magnesium
  • Calcium

The ingredients for the therapy are adjusted specifically to match each patient’s unique needs. Two of the most popular vitamin injections are Vitamin B12 (an important component of cell reproduction, blood formation, and protein or tissue synthesis) and Lipotropic injections (to help encourage weight loss).

Why Do We Need Vitamin Supplements at All?

We all know that vitamins are critical for good health. In vitamin deficient countries – even our own – a lack of key vitamins can lead to bone loss or fractures, chronic infections, and even neurologic symptoms. But with the fortification of foods with vitamins, true vitamin deficiencies are rare. Does this mean that we still don’t need extra vitamin supplementation?

This is where it gets tricky: in the perfect world with the perfect body and immune system, a person would only need the natural fruits and vegetables that nature designed for us – I sometimes refer to this as the Biblical diet that Adam and Eve had in the Garden of Eden.

But in our real world, most of us have been at the disadvantage of getting nutrients that have been extensively processed and so are devoid of the full array of vitamins and minerals.

As a result, when we develop illnesses, our body’s immune system isn’t at full capacity to combat infections or correct immune imbalances.

Eating foods that contain high doses of natural vitamins and minerals is a great start, but ingested vitamins must go through the harsh environment of the digestive tract, which breaks them down and decreases their absorption rate to only 30%. This percentage rate can be even less if you are taking acid blockers or have digestive problems.

The body depends on receiving vitamins and minerals from the diet because it cannot make them itself. IV vitamin therapy helps if your body is low in vitamins and minerals due to not consuming the proper foods, or your body is unable to metabolize them correctly.

Terms of service.

You can find our terms of service here.


Private Complementary and Alternative Healthcare clinic.
1st Floor
185 Tower Bridge Road
United Kingdom
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